| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.50522975 |
| By utilizing XIAP mutants , we now report that XIAP binds to the ' native ' apoptosome complex via a specific interaction with the small p 12 subunit of processed caspase 9 . 0.50522975^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Coexpression of the catalytically inactive , dominant negative , mutant caspase 9 , XIAP , or treatment with the caspase inhibitor , zVAD , significantly inhibited the increase in apoptosis of HL 60 / Apaf 1 cells ( P < 0 . 01 ) . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| The prominent decrease of 10 chromosome linked inhibitory apoptosis protein ( XIAP ) in the cytosol may explain the activation of caspase 9 induced by RA and BMP 4 treatment . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| This conclusion is supported by the observation that in HL 60 / Apaf 1 cells , ectopic expression of dominant negative caspase 9 , its inhibitory short isoform caspase 9b , or XIAP or treatment with the caspase inhibitor zVAD ( 50 microM ) inhibited Apaf 1 induced caspase 8 and Bid cleavage , mitochondrial deltapsim , release of cyt c , and apoptosis . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| XIAP regulates DNA damage induced apoptosis downstream of caspase 9 cleavage . ^^^ In addition , we show that ara C induces the association of XIAP with the cleaved fragments of caspase 9 and thereby inhibition of caspase 9 activity . ^^^ These results demonstrate that XIAP functions downstream of procaspase 9 cleavage as an inhibitor of both proteolytically processed caspase 9 and 3 in the cellular response to genotoxic stress . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Here we show that the Bir 3 domain is the minimal region of XIAP that is needed for potent caspase 9 inhibition . ^^^ Based on site directed mutagenesis , we have identified the regions of the Bir 3 domain of XIAP that are important for inhibiting caspase 9 . ^^^ These results suggest that XIAP inhibits caspase 3 and caspase 9 in a different manner . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Immunoblot analyses showed that bone marrow precursors express appreciable levels of caspase 3 and caspase 9 but no or very low levels of c fms ( M CSF receptor ) and the apoptosis regulators 10 linked inhibitor of apoptosis protein ( XIAP ) , c IAP 1 , Bcl 2 and Bax . ^^^ Unlike bone marrow precursor cells , mature BMDM ( day 7 10 ) were resistant to apoptosis induced by M CSF depletion , which includes the activation of caspase 3 and caspase 9 and the degradation of XIAP , Bcl 2 and Bax proteins in the process . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Interestingly , overexpression of caspase 9S and 10 linked inhibitor of apoptosis ( XIAP ) , both able to inhibit caspase 9 activity , failed to block apoptosis . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| DIABLO promotes apoptosis by removing MIHA / XIAP from processed caspase 9 . ^^^ Here , we show that after UV radiation , MIHA prevented apoptosis by inhibiting caspase 9 and caspase 3 activation . ^^^ Unlike Bcl 2 , MIHA functioned after release of cytochrome c and DIABLO from the mitochondria and was able to bind to both processed caspase 9 and processed caspase 3 to prevent feedback activation of their zymogen forms . ^^^ Once released into the cytosol , DIABLO bound to MIHA and disrupted its association with processed caspase 9 , thereby allowing caspase 9 to activate caspase 3 , resulting in apoptosis . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Introduction of these mutations into full length XIAP reduced caspase 3 inhibitory activity up to 500 fold , but did not affect its ability to inhibit caspase 9 or interact with the IAP antagonist DIABLO . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| To identify human proteins that bind to the Smac and caspase 9 binding pocket on the baculoviral inhibitor of apoptosis protein ( IAP ) repeat 3 ( BIR 3 ) domain of human XIAP , we used BIR 3 as an affinity reagent , followed by elution with the BIR 3 binding peptide AVPIA , microsequencing , and mass spectrometry . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| However , other proteins analyzed [ namely , Apaf 1 , Bax , Bid , caspase 3 , caspase 8 , caspase 9 , 10 linked inhibitor of apoptosis protein ( XIAP ) , cellular inhibitor of apoptosis protein ( cIAP ) 1 , cIAP 2 , cytochrome c , Fas , Fas ligand , FLIP , p 53 , and poly ( ADP ribose ) polymerase ] were not affected by either rituximab or cisplatin . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| The anti apoptotic activity of XIAP is retained upon mutation of both the caspase 3 and caspase 9 interacting sites . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| SN 50 induced apoptosis in MM cell lines and patient cells ; down regulated expression of Bcl 2 , A 1 , 10 chromosome linked inhibitor of apoptosis protein ( XIAP ) , cellular inhibitor of apoptosis protein 1 ( cIAP 1 ) , cIAP 2 , and survivin ; up regulated Bax ; increased mitochondrial cytochrome c release into the cytoplasm ; and activated caspase 9 and caspase 3 , but not caspase 8 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| The inhibitory potency of acetyl Asp Glu Val Asp aldehyde ( ' Ac DEVD CHO ' ) , benzyloxycarbonyl Val Ala Asp fluoromethylketone ( ' Z VAD FMK ' ) and the endogenous caspase inhibitor 10 chromosome linked inhibitor of apoptosis protein ( ' XIAP ' ) against recombinant caspase 9 were predictive of the efficacy of these compounds in a cell free system . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Finally , exogenous glutathione protected T cells from thimerosal induced apoptosis by upregulation of XIAP and cIAP 1 and by inhibiting activation of both caspase 9 and caspase 3 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| CDDO induced apoptosis of CLL B cells was blocked by cytokine response modifier A ( CrmA ) , a suppressor of caspase 8 , but not by 10 linked inhibitor of apoptosis protein baculovirus IAP repeat 3 ( XIAP BIR 3 ) , a fragment of XIAP , which selectively inhibits caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Regulation of the Apaf 1 / caspase 9 apoptosome by caspase 3 and XIAP . ^^^ In addition to the PEPD site , caspase 9 contains a caspase 3 cleavage site ( DQLD ( 330 ) ) , which when cleaved , produces a smaller p 10 subunit in which the NH ( 2 ) terminal 15 amino acids of p 12 , including the XIAP BIR 3 binding motif , are removed . ^^^ In addition , we characterized the effect of caspase 3 feedback cleavage of caspase 9 on the rate of caspase 3 activation , and the potential ramifications of Asp ( 330 ) cleavage on XIAP mediated inhibition of the apoptosome . ^^^ In addition , cleavage at Asp ( 330 ) exposed a novel p 10 NH ( 2 ) terminal peptide motif ( AISS ) that retained the ability to mediate XIAP inhibition of caspase 9 . ^^^ Thus , whereas feedback cleavage of caspase 9 by caspase 3 significantly increases the activity of the apoptosome , it does little to attenuate its sensitivity to inhibition by XIAP . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Mechanism of XIAP mediated inhibition of caspase 9 . ^^^ This paper reports the crystal structure of caspase 9 in an inhibitory complex with the third baculoviral IAP repeat ( BIR 3 ) of XIAP at 2 . 4 A resolution . ^^^ Thus , XIAP sequesters caspase 9 in a monomeric state , which serves to prevent catalytic activity . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| These results were explained by the discovery of a simultaneous Stx 1 dependent increase in endogenous XIAP , a direct inhibitor of caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Western blot analysis confirms that Smac induced apoptosis proceeds via a pathway mediated primarily by caspase 9 that can be inhibited by zLEHD fmk and overexpression of the 10 linked inhibitor of apoptosis protein ( XIAP ) . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| This lack of caspase 9 activation might be related to an impaired ' crosstalk ' with the caspase 8 pathway as suggested by the missing Bid cleavage and to the appearance of an XIAP cleavage product known to inhibit caspase 9 activation . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Here we show that codelivery of DNA encoding inhibitors of apoptosis ( BCL xL , BCL 2 , XIAP , dominant negative caspase 9 , or dominant negative caspase 8 ) with DNA encoding model antigens prolongs the survival of transduced DCs . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| To clarify this issue , the authors examined time dependent expression and interaction among XIAP , Smac / DIABLO , and activated caspase 9 by immunohistochemistry , Western blot analysis , and immunoprecipitation using an in vivo transient focal cerebral ischemia model . ^^^ XIAP increased concurrently with the release of Smac / DIABLO and the appearance of activated caspase 9 during the early period after reperfusion injury . ^^^ The bindings of XIAP to Smac / DIABLO and to caspase 9 and the binding of Smac / DIABLO to caspase 9 reached a peak simultaneously after transient focal cerebral ischemia . ^^^ These results suggest that the XIAP pathway was activated upstream of the caspase cascade and that interaction among XIAP , Smac / DIABLO , and caspase 9 plays an important role in the regulation of apoptotic neuronal cell death after transient focal cerebral ischemia . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| The inhibitor of apoptosis proteins ( IAP ) are endogenous caspase inhibitors in the metazoan and characterized by the presence of baculoviral IAP repeats ( BIR ) . 10 linked IAP ( XIAP ) contains three BIR domains and directly inhibits effector caspases such as caspase 7 via a linker _ BIR2 fragment and initiator caspases such as caspase 9 via the BIR 3 domain . ^^^ Here , using glutathione S transferase pulldown and caspase activity assay , we show that Smac is ineffective in relieving either caspase 7 or caspase 9 inhibition by XIAP domain fragments . ^^^ However , Smac is effective in removing caspase 7 and caspase 9 inhibition by XIAP fragments containing both the BIR 2 and BIR 3 domains . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Smac 3 disrupts processed caspase 9 binding to XIAP , promotes caspase 3 activation , and potentiates apoptosis . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| In H 460 cells , the overexpressed XIAP , but not c IAP 1 , bound to the processed form of caspase 9 and suppressed the activation of downstream effector caspases . ^^^ Moreover , the defect in apoptosome activity in H 460 cells was dramatically restored by the IAP targeting SmacN 7 peptide , which disrupted XIAP caspase 9 binding , indicating an essential role of the IAP in the apoptosome inhibition . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Exogenous expression of XIAP is sufficient to inhibit caspase 9 activation , Smac release , and cell death induced by etoposide . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| When the extract was activated by cytochrome c , caspase 3 recruitment to the apoptosome was not observed , although apoptotic protease activating factor 1 ( Apaf 1 ) , caspase 9 , and 10 linked inhibitor of apoptosis protein ( XIAP ) existed in the apoptosome . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| To elucidate the role of caspase 9 in ALS , we examined the effects of an inhibitor of 10 chromosome linked inhibitor of apoptosis ( XIAP ) , a mammalian inhibitor of caspase 3 , 7 and 9 , and p 35 , a baculoviral broad caspase inhibitor that does not inhibit caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| In comparison with Smac peptides targeting XIAP mediated caspase 9 inhibition , which do not directly induce cell death , it appears that liberating downstream caspases rather than upstream caspases may be a preferred strategy for cancer drug discovery . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Caspase inhibition by overexpression of XIAP prevented caspase 9 and caspase 3 activation but not cell death , presumably because of increased caspase independent cell death . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Interestingly , and in contrast to the 10 chromosome linked inhibitor of apoptosis protein ( XIAP ) , NAIP mediated inhibition of caspase 9 was not countered by a peptide containing an amino terminal IAP binding motif ( IBM ) . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Here we describe the establishment of Jurkat derived cell lines stably overexpressing either full length XIAP or a truncation mutant of XIAP that can only inhibit caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| FTY 720 selectively induced cell apoptosis in hepatoma cell lines with overexpression of cleaved caspase 3 and caspase 9 , but the same phenomena were not found in MIHA cells . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Designing small molecule inhibitors that target the BIR 3 domain of XIAP , where Smac / DIABLO ( second mitochondria derived activator of caspase / direct IAP binding protein with low pI ) and caspase 9 bind , is a promising strategy for inhibiting the antiapoptotic activity of XIAP and for overcoming apoptosis resistance of cancer cells mediated by XIAP . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| In the cytoplasm , survivin inhibits apoptosis by interacting with caspase 9 in the presence of the HBXIP cofactor , by binding to Smac or associating with XIAP . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Active phenylurea based compounds dissociated effector protease caspase 3 but not initiator protease caspase 9 from XIAP in vitro and restored caspase 3 but not caspase 9 enzymatic activity . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Transient transfection of Nodal and ALK 7 ca significantly decreased 10 linked inhibitor of apoptosis protein ( Xiap ) expression , activated both caspase 3 and caspase 9 , and increased apoptosis as determined by Hoechst nuclear staining and flow cytometry . ^^^ These findings demonstrate that Nodal induces apoptosis and inhibits proliferation via ALK 7 and Smad2 / 3 and that the effect of Nodal ALK 7 on apoptosis may be mediated in part by the down regulation of Xiap and activation of caspase 9 and caspase 3 . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Expression levels of Bcl 2 and 10 linked inhibitor of apoptosis ( XIAP ) were used to determine the status of the caspase 9 mediated pathway . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| A tissue specific homologue of XIAP , known as ILP 2 ( IAP like protein 2 ) , has previously been implicated in the control of apoptosis in the testis by direct inhibition of caspase 9 . ^^^ Fusion of a 9 residue linker from XIAP to the N terminus of ILP 2 restored tight caspase 9 inhibition , dramatically increased conformational stability and allowed crystallization of the ILP 2 BIR domain in a form strikingly similar to the XIAP third BIR domain . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| In examining another regulator of cleaved caspase 9 , 10 chromosome linked inhibitor of apoptosis protein ( XIAP ) , we observed that TNF alpha treatment induced fragmentation of XIAP , which was also enhanced by the PI3K inhibitor . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Furthermore , treatment with ucf 101 before reperfusion attenuated 10 linked inhibitor of apoptosis protein degradation and inhibited caspase 9 and caspase 3 activities . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Baicalein inhibited the phosphorylation of IkB alpha , which was followed by decreased expression of the IL 6 and XIAP genes and activation of caspase 9 and caspase 3 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| The XIAP antagonist 1396 12 induced activation of downstream caspases 3 and 7 prior to the activation of upstream caspase 8 and caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Here we show that 10 linked IAP ( XIAP ) mediates the polyubiquitination of caspase 9 and Smac . ^^^ The large subunit of mature caspase 9 was polyubiquitinated by XIAP in vitro , while procaspase 9 was not . ^^^ Furthermore , the polyubiquitinated form of caspase 9 accumulated in an XIAP dependent manner in intact cells . ^^^ The ubiquitination of caspase 9 was significantly inhibited in the presence of mature Smac , whereas XIAP was also found to promote the polyubiquitination of cytosolic Smac both in vitro and in intact cells . ^^^ These findings suggest that XIAP functions as ubiquitin ligase toward mature caspase 9 and Smac to inhibit apoptosis . . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| During apoptosis , a key event is the release of Smac / DIABLO ( an inhibitor of XIAP ) and cytochrome c ( Cyt c , an activator of caspase 9 ) from mitochondria to cytosol . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Ellipticine increased the expression of Bax , but decreased the level of Bcl 2 , Bcl XL and 10 linked inhibitor of apoptosis protein ( XIAP ) , and subsequently triggered the mitochondrial apoptotic pathway ( release of cytochrome c , and activation of caspase 9 and 3 ) . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Lastly , we show that NF kappaB inhibits c Myc induced activation of caspase 9 and caspase 3 through up regulation of the antiapoptotic target genes Bcl 10 ( L ) and 10 linked inhibitor of apoptosis ( XIAP ) . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Apoptosis by I3C involves downregulation antiapoptotic gene products , including Bcl 2 , Bcl xL , survivin , inhibitor of apoptosis protein ( IAP ) , 10 chromosome linked IAP ( XIAP ) , and Fas associated death domain protein like interleukin 1 beta converting enzyme inhibitory protein ( FLIP ) ; upregulation of proapoptotic protein Bax ; release of micochondrial cytochrome C ; and activation of caspase 9 and caspase 3 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| In SNU 251 cells expressing BRCA 1 ( S1423 / 1524A ) , the inhibitory interaction between 10 linked inhibitor of apoptosis protein ( XIAP ) and caspase 9 remains intact after UV , compared with cells expressing wt BRCA 1 . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| In lysates containing very low levels of Smac and Omi / HtrA2 , XIAP ( 10 linked inhibitor of apoptosis ) binds tightly to caspase 9 in the apoptosome complex , and as a result caspase 7 processing is abrogated . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| FGF 1 attenuated the HI induced increase in activated caspase 3 , caspase 9 and cleaved PARP protein levels and markedly blocked the HI induced decrease in XIAP expression under the conditions at which FGF 1 showed significant neuroprotection . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| The damaged and surviving neurons in hippocampus were observed by terminal deoxynucleotidyl transferase mediated dUTP nick end labeling ( TUNEL ) and cresyl violet staining , the expression of caspase 8 , Bid , XIAP , caspase 9 , cytochrome c and Smac / DIABLO were detected with immunofluorescence and Western blot . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| Likewise , A . phagocytophilum infection inhibited the pro apoptotic Bax translocation to mitochondria , activation of caspase 9 , the initiator caspase in the intrinsic pathway , and the degradation of a potent caspase inhibitor , 10 chromosome linked inhibitor of apoptosis protein ( XIAP ) , during spontaneous neutrophil apoptosis . ^^^ |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P98170 |
Pubmed |
SVM Score :0.0 |
| NA |
|