| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The kinase Akt and p 21 Ras , an Akt activator , induced phosphorylation of pro caspase 9 ( pro Casp 9 ) in cells . ^^^ Cytochrome c induced proteolytic processing of pro Casp 9 was defective in cytosolic extracts from cells expressing either active Ras or Akt . ^^^ Akt phosphorylated recombinant Casp 9 in vitro on serine 196 and inhibited its protease activity . ^^^ Mutant pro Casp 9 ( Ser196Ala ) was resistant to Akt mediated phosphorylation and inhibition in vitro and in cells , resulting in Akt resistant induction of apoptosis . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The down regulated genes were TRAIL , TNFR , AP 13 , IGFBP 3 , SARP 3 , PKB , IGFBP , CASP 7 , CASP 9 , TNFSF 6 , TRICK2A , CAS , TRAIL R 2 , RATS 1 , hTRIP , TNFb and TNFRSF 5 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Survival factors can suppress apoptosis in a transcription independent manner by activating the serine / threonine kinase Akt , which then phosphorylates and inactivates components of the apoptotic machinery , including BAD and Caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| These data suggest that M CSF may induce cell survival through Akt induced suppression of caspase 9 activation . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The phosphatase inhibitors orthovanadate and okadaic acid prevented catalytic processing and activation of caspase 3 and digestion of Akt and partially inhibited cleavage of caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Akt phosphorylation site found in human caspase 9 is absent in mouse caspase 9 . ^^^ Caspase 9 is one caspase upstream of caspase 3 and its activation is stimulated by Apaf 1 / cytochrome c and inhibited by Akt signals . ^^^ Recently , it was shown that human caspase 9 is phosphorylated by Akt and that its protease activity is reduced . ^^^ Curiously , the Akt phosphorylation sites and motifs found in human caspase 9 were absent in both mouse caspase 9L and 9S . ^^^ Mouse caspase 9 was not phosphorylated by activated Akt in vitro . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Akt has been shown to mediate its apoptosis suppressive effects via phosphorylation of Bad or caspase 9 . ^^^ Mutation of the Akt site within caspase 9 significantly reduced the apoptosis suppressive effect of insulin . ^^^ In endothelial cells , Akt seems to mediate its antiapoptotic effect , at least in part , via phosphorylation of caspase 9 rather than Bad . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Addition of cytochrome c and dATP to lysates from cells expressing active Akt do not activate caspase 9 or 3 and immunoprecipitated Akt added to control lysates blocks cytochrome c induced activation of the caspase cascade . ^^^ Taken together , these data suggest that Akt inhibits activation of caspase 9 and 3 by posttranslational modification of a cytosolic factor downstream of cytochrome c and before activation of caspase 9 . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Adhesion of integrin receptors to extracellular matrix activates a survival signaling pathway in epithelial cells where Akt phosphorylates and blocks the activity of proapoptotic proteins such as the BCL 2 family member Bad , the forkhead transcription factor FKHRL 1 , and caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Decreased phosphatidylinositol 3 kinase and Akt activities occurred in association with cytochrome c release and caspase 9 activation when cells were placed in defined medium . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| CEP treatment induced activation of caspase 9 and 3 accompanied by cleavage of PARP , Bid , lamin B 1 , and DFF45 / ICAD in both Jurkat and K 562 cells , whereas caspase 8 activation and Akt cleavage were observed only in Jurkat cells . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The mechanism of GT1b activation of caspase 9 involved inhibition of beta ( 1 ) integrin serine / threonine phosphorylation and decreased phosphorylation of both integrin linked kinase and protein kinase B / Akt at its Ser 473 site , leading to cytochrome c release from mitochondria . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Wortmannin or LY 294002 , two structurally dissimilar inhibitors of phosphatidylinositol 3 ' kinase ( PI 3 kinase ) , abolished both cisplatin induced Akt phosphorylation and Bad phosphorylation , and promoted cisplatin induced early and accelerated activation of caspase 3 and caspase 9 , but not of caspase 8 and caspase 1 , indicating that inhibition of the Akt / PKB phosphorylation pathway enhances the mitochondrial dependent activation of caspases . ^^^ CONCLUSIONS : These studies demonstrate differential activation and role of caspases in cisplatin injury , and provide the first evidence of cisplatin induced induction of the Akt / PKB phosphorylation pathway , inhibition of which enhances activation of caspase 3 and caspase 9 . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| After phosphorylation , activated Akt inactivates other apoptogenic factors , such as Bad or caspase 9 , thereby inhibiting cell death . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Diminished Akt signaling promoted the cleavage of caspase 3 , and both caspase 3 cleavage and FasL induction were inhibited by transduction of dominant negative caspase 9 or the caspase 8 inhibitor CrmA . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Because other cytokines and bacterial cell wall products also induce monocyte survival , we hypothesized that these factors may also suppress caspase 3 and caspase 9 activation and activate Akt in human monocytes . ^^^ These results suggest that monocyte survival factors may suppress DNA fragmentation , caspase 9 , and caspase 3 activation in a PI 3 kinase dependent manner , perhaps through the activation of Akt . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| PI 3 K downstream signaling events were evaluated by measuring the expression of cytosolic total and phosphorylated Akt and Bad proteins by Western blot analyses , and caspase 9 activity . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Once activated , Akt can prevent or delay apoptosis by phosphorylation dependent inhibition or activation of multiple signaling molecules involved in apoptosis , such as BAD , caspase 9 , GSK 3 , and NF kappaB and forkhead family transcription factors . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Ganglioside depletion promotes survival of the human keratinocyte derived SCC 12 cell line through upregulated phosphorylation of beta 1 integrin , and increased phosphorylation and activity of integrin linked kinase , protein kinase B / Akt , and Bad , with resultant inhibition of caspase 9 activation . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Hepatocyte apoptosis induced by glycodeoxycholate ( GCDC ) was associated with mitochondrial depolarization , activation of caspases , the release of cytochrome c from the mitochondria , and translocation of BAX from the cytosol to the mitochondria . cAMP inhibited GCDC induced apoptosis , caspase 3 and caspase 9 activation , and cytochrome c release in a PI3K dependent manner . cAMP activated PI3K in p 85 immunoprecipitates and resulted in PI3K dependent activation of the survival kinase Akt . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Potentiation of Akt and suppression of caspase 9 activations by electroacupuncture after transient middle cerebral artery occlusion in rats . ^^^ In the current study , we investigated the effects of EA treatment on the activations of survival Akt and proapoptotic caspase 9 after 90 min of transient middle cerebral artery occlusion ( tMCAO ) in rat . ^^^ These results suggest that EA potentiated the Akt and suppressed the caspase 9 activations , and may have a potential to reduce the number of neuronal cells undergoing apoptotic cell death . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Activation of Akt maintains Bad phosphorylation and prevents its binding to mitochondrial targets , decreases caspase 9 activity , and prevents the translocation of forkhead transcription factors ( FKHR ) . ^^^ Akt activation ; increase in islet viability ; and decrease in Bad phosphorylation , cytochrome release , caspase 9 activation , and translocation of FKHR were observed after simvastatin treatment , effects reversed by LY 294002 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| As well as inhibiting the Akt NFkappaB pathway , InsP ( 6 ) itself caused mitochondrial permeabilization , followed by cytochrome c release , which later caused activation of the apoptotic machinery , caspase 9 , caspase 3 and poly ( ADP ribose ) polymerase . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Several end points of cytokine action including decreases in phospho CREB , phospho Akt , and BCl 2 levels and activation of caspase 9 were observed in isolated mouse islets . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| After phosphorylation , activated Akt inactivates BAD or caspase 9 or other apoptogenic components , thereby inhibiting cell death . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Immunoreactivities of phosphorylated Akt ( p Akt ) , cleaved caspase 9 ( c cas 9 ) , and 3 ( c cas 3 ) increased after the reperfusion in the penumbra in vehicle group with peaks at 3 h , 8 h , and 1 day , respectively . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| In extending these findings , we now report a marked decrease in protein kinase C ( PKC ) isoforms , reduced Akt serine / threonine kinase activity , Bcl 2 associated death promoter ( BAD ) phosphorylation and enhanced p 38 mitogen activated protein kinase ( MAPK ) and caspase 9 and 3 activation , 12 h after addition of both 5 micro m Abeta and 5 micro m Fe2+ . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Furthermore , in the presence of CVB 3 infection , the intensity of the phosphorylation of Akt was further enhanced and associated with a delayed activation of caspase 9 and caspase 3 . ^^^ Moreover , IGTP expression inhibited viral replication and delayed CVB 3 induced cleavage of eukaryotic translation initiation factor 4G , indicating that IGTP mediated cell survival relies on not only the activation of PI 3 K / Akt , inactivation of GSK 3 and suppression of caspase 9 and caspase 3 but also the inhibition of viral replication . . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Inhibition of UV induced activation of Akt increased release of mitochondrial cytochrome c 3 . 5 fold , and caused appearance of active forms of caspase 9 , caspase 8 , and caspase 3 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Furthermore , expression of myristoylated Akt 1 not only prevented cell injury through the prevention of membrane phosphatidylserine exposure and genomic DNA fragmentation but also inhibited microglial activation and proliferation that required the inhibition of caspase 9 , caspase 3 , and caspase 1 like activities linked to cytochrome c release . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Furthermore , ADR induced activation of caspase 9 , a downstream molecule of Akt , was inhibited for at least 24 h after HGF / SF stimulation , but it was not affected by EGF stimulation . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Adenoviral mediated increased in IRS 2 protein levels protected against fatty acid ( FFA ) induced apoptosis , associated with increased activation of PKB and decreased levels of activated caspase 9 . ^^^ This was associated with decreased activation of PKB and increased caspase 9 activation . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Downstream targets of PI3K AKT include the proapoptotic protein BAD , caspase 9 , NF kappaB , and Forkhead . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| We found that the expression of mitochondrial apoptosis related genes ( Bcl 2 associated protein 10 , BAX ; apoptotic protease activating factor 1 , Apaf 1 ; Caspase 9 and serine / threonine protein kinase , PKB ) is elevated in Trichinella spiralis infected muscles during encapsulation . ^^^ Immunocytochemistry revealed that pro apoptosis factor ( BAX , Apaf 1 and Caspase 9 ) are predominantly expressed in the basophilic cytoplasm ( infected muscle cell origin ) and anti apoptosis factor ( PKB ) in the eosinophilic cytoplasm ( satellite cell origin ) of the nurse cell . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Cell cycle distribution and cell cycle regulator proteins p 27 ( Kip 1 ) and cyclin D 1 , together with the PI 3 K / Akt pathway , mitogen activated protein kinases and cleaved caspase 3 and caspase 9 , were evaluated . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| These results suggest the sequential mechanism of diclofenac induced apoptosis of HL 60 cells : ROS generation suppresses Akt activity , thereby activating caspase 8 , which stimulates Bid cleavage and induces cytochrome c release and the activation of caspase 9 and 3 in a CsA insensitive mechanism . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Inactivation of Akt was associated with dephosphorylation of BAD , increased cytochrome c release , and activation of caspase 3 and caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| In this study , we compared the potency of alpha TS and alpha TOB using a panel of bioassays : cell growth , TUNEL labelling for apoptosis , PARP cleavage , caspase 3 and caspase 9 activation , as well as Akt and JNK phosphorylation . ^^^ Nonetheless , these two compounds shared the same mechanism of targeting the Akt and JNK signaling pathways , and activating the intrinsic cell death mediators of caspase 9 and caspase 3 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Furthermore , binding to these receptors triggers the phosphatidyloinositol 3 kinase ( PI3K ) / Akt signal transduction system and , in consequence , leads to the inhibition of PCD by activating antiapoptotic proteins through the transcription factor NFkappaB and inhibiting proapoptotic signaling by Bad , caspase 9 , caspase 3 , and other effectors . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Examined apoptosis related genes included tumor suppressor genes p 53 , p 53 ; mouse double minute 2 , MDM 2 ; cyclin dependent kinase inhibitor p 21 ( WAF 1 ) , p 21 ( waf ) ; Bcl 2 associated protein 10 , BAX ; apoptotic protease activating factor 1 , Apaf 1 ; Caspase 9 and serine / threonine protein kinase , PKB . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Proteins for Western blot analysis included Fas associated death domain ( FADD ) and caspase 8 for extrinsic pathway , as well as Bcl 2 , Bcl XL , Bax , Bad , Bid , Akt , p Akt , and caspase 9 for intrinsic pathway . ^^^ Levels of FADD , Bax , Bad , and Bid were substantially increased in the TA induced myopathy group , whereas Bcl 2 , Bcl XL , Akt , p Akt , and caspase 9 did not change between control and myopathy groups . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| ZD 1839 inhibited the phosphorylation of Akt , and enhanced TRAIL induced apoptosis via activation of caspase 3 and caspase 9 , and inactivation of Bcl xL . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| TRAIL induced caspase 8 activation , Bid processing , drop of DeltaPsi ( m ) , and poly ADP ribose polymerase ( PARP ) cleavage but not in caspase 9 activation , and these events were inhibited in HER2 / PI3K / Akt suppressed BT 474 cells , which on the other hand exhibited downregulation of Bcl xL and increased response to mitomycin C . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| To ascertain the mechanism of epithelial apoptosis , we determined that PAR 1AP , thrombin , and leukocyte elastase dissipated mitochondrial membrane potential , induced translocation of cytochrome c to the cytosol , enhanced cleavage of caspase 9 and caspase 3 , and led to JNK activation and Akt inhibition . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Other Akt downstream targets , BAD , caspase 9 and the Forkhead transcription factor ( FKHR ) , were not phosphorylated during the course of infection by EV 71 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The Akt inhibitor QLT 394 also demonstrated antiproliferative effects in a dose and time dependent manner , dephosphorylated ribosomal S 6 and cleaved caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The phosphorylated Akt and activated fragments of caspase 9 , caspase 3 were examined by Western blotting analysis . ^^^ Western blotting analysis showed remarkable activation of caspase 9 , caspase 3 and dephosphorylation of Akt ( Thr ( 308 ) ) . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| Pyrrolidine dithiocarbamate inhibition of luteolin induced apoptosis through up regulated phosphorylation of Akt and caspase 9 in human leukemia HL 60 cells . ^^^ Therefore , not only was PDTC able to stop the apoptosis of HL 60 cells treated with luteolin , it was also found to increase phosphorylation of Akt and caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| At the cellular level , ischemia hypoxia produced proinflammatory cytokines and simultaneously activated both prosurvival ( eg , synthesis of heat shock 70 protein , phosphorylation of ERK and AKT ) and proapoptosis signaling pathways ( eg , release of cytochrome c and AIF from mitochondria , cleavage of caspase 9 and 8 ) . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| The heat shock protein 90 inhibitor 17 AAG induces cell cycle arrest and apoptosis in mantle cell lymphoma cell lines by depleting cyclin D 1 , Akt , Bid and activating caspase 9 . ^^^ |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P55211 and P31749 |
Pubmed |
SVM Score :0.0 |
| NA |
|