Pubmed abstracts for Protein-Protein Interaction search result :

Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Compared to membranes from unaffected canines , those from the kidney inner medulla of NDI dogs possessed normal V 2 receptor numbers , but with 10 fold lower affinity for [ Arg 8 ] vasopressin ( AVP ) . ^^^ Prolonged treatment with V 2 agonists , 1 deamino [ D Arg 8 ] VP ( dDAVP ) and 1 deamino [ Val 4 , Sar 7 ] AVP ( dVSAVP ) , rendered the NDI affected dogs near normal in terms of water intake and urine osmolality . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
We conclude that failure by AVP to increase cAMP in cells of collecting ducts , which results from anomalously high activity of one or two specific isozymes of cAMP PDE , is the major or sole cause for the excretion of hypotonic urine in NDI mice ( DI + / + Severe strain ) . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
In the NDI group , the 10 ( 6 ) M AVP stimulated cAMP contents failed to increase completely , and the levels were significantly lower than that of the control group ( 10 . 4 + / 1 . 4 vs . 48 . 4 + / 4 . 7 fmol / mm , P less than 0 . 001 ) . ^^^ Pretreatment with pertussis toxin ( PT ) , an inhibitor of inhibitory G protein ( Gi ) , did not affect the basal cAMP levels in both groups , although it increased AVP stimulated cAMP production in the NDI group in a dose and time dependent manner . ^^^ AVP stimulated cAMP production with over 100 ng / ml PT in the NDI group reached the levels observed in the control group . ^^^ From these results it is suggested that Li impairs AVP sensitive AdC not through inhibition of Gs but through activation of Gi and that PGE 2 may not be involved in the cellular pathogenesis of NDI at least in the rat at the step of cAMP formation . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
V 2 receptor binding activity and induction of cAMP production in response to [ Arg 8 ] vasopressin ( AVP ) were exhibited by all cell lines carrying the wild type NDI locus , in contrast to control cell lines . ^^^ The V 2 specific agonist [ Mpa 1 , Val 4 , Sar 7 ] AVP was as potent as AVP in inducing cAMP production by NDI DNA carrying cells , whereas no response was shown to other hormones such as calcitonin , oxytocin ( less than 10 ( 8 ) M ) , isoproterenol , or an oxytocin specific agonist . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
In mice with hereditary nephrogenic diabetes insipidus ( NDI ) , the high activity of cAMP phosphodiesterase ( cAMP PDIE ) in medullary collecting tubules ( MCT ) prevents the increase in cAMP content in response to vasopressin [ Arg 8 ] vasopressin ( AVP ) . ^^^ Even when the cAMP response to AVP is partly corrected by cAMP PDIE inhibitor 1 methyl 3 isobutylxanthine ( MIX ) , under all tested conditions the cAMP levels in MCT of NDI mice remained much lower than in controls ( B . ^^^ In the presence of 1 microM AVP and 0 . 05 mM MIX , the cAMP levels accumulated in MCT of NDI mice were four times lower compared with controls , but the levels of ATP and NAD were not different . ^^^ Although AVP alone had little effect on cAMP levels in mouse MAL in the presence of 0 . 1 mM forskolin , the AVP elicited a 20 fold increase of cAMP of both the control and NDI mice . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
To understand the mechanisms of AVP resistance underlying this disorder , we have analyzed the vasopressin V 2 receptor gene in two unrelated Japanese kindreds with NDI and expressed the mutants to characterize their functional properties . ^^^ Nephrogenic diabetes insipidus ( NDI ) is a rare 10 linked disorder associated with renal tubule resistance to arginine vasopressin ( AVP ) . ^^^ Our results suggest an introduction of a new cysteine residue in the extracellular domain and a receptor truncation removing one third of the carboxyl terminus could impair ligand binding activity of the V 2 receptor through a post transcriptional mechanism , thereby causing AVP resistance in the NDI patients . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Following the recent characterization of the cDNA and genomics sequences encoding the human V 2 receptor to AVP ( AVPR 2 ) , 10 linked NDI has been found to be due to mutations in the AVPR 2 gene that maps to the chromosome Xq 28 region . ^^^ Congenital nephrogenic diabetes insipidus ( NDI ) is a rare inherited disorder characterized by the inability of the kidney to concentrate urine in response to vasopressin ( AVP ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
To determine if the AVP induced vasodilation in human forearm vessels is mediated by the V 2 receptor , we examined the effects of OPC 31260 ( a novel vasopressin V 2 receptor antagonist ) on AVP induced vasodilation . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Responses were compared with those of arginine vasopressin ( AVP ) and vasopressin V 2 receptor stimulation resulting from infusion of a V 1 receptor antagonist with AVP . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nephrogenic diabetes insipidus ( NDI ) is characterized by a resistance of the kidney towards arginine vasopressin ( AVP ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Based on these results , we conclude that at least two factors play an important role in the pathogenesis of NDI consequent to chronic oral administration of Li : ( a ) decreased ability of MCT and PCD to generate and accumulate cAMP in response to stimulation by AVP ; this defect is primarily due to diminished activity of AdC in these tubular segments caused by prolonged exposure to Li ; and ( b ) lower osmolality of renal papillary tissue , due to primarily to depletion of urea , which decreases osmotic driving force for water reabsorption in collecting tubules . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
In one NDI patient , arginine vasopressin ( AVP ) was given in incremental doses ( 62 . 5 4000 pg / kg / min ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nephrogenic diabetes insipidus ( NDI ) is a rare 10 linked disorder exhibiting renal resistance to the antidiuretic action of arginine vasopressin ( AVP ) . ^^^ Our results suggest that different V 2 receptor defects could be responsible for AVP resistance in individual NDI kindreds . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Mice and patients with NDI have evidence of increased AVP synthesis . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The value of a water deprivation test incorporating urinary arginine vasopressin ( AVP ) measurement was investigated in 13 patients with polydipsia and / or polyuria ( complete central diabetes insipidus [ CCDI ] in four ; incomplete central diabetes insipidus [ ICDI ] in five ; secondary nephrogenic diabetes insipidus [ NDI ] in three ; compulsive water drinking [ CWD ] in one ) and a group of 25 control subjects ( C ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The studies of animal models of nephrogenic diabetes insipidus ( NDI ) suggest that abnormally high activity of cAMP phosphodiesterase ( cAMP PDE ) , may cause unresponsiveness to the diuretic effect of AVP . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
While many studies have demonstrated a nephrogenic diabetes insipidus syndrome ( NDI ) with prolonged lithium ( Li ) treatment , experiments in the isolated rat papillary collecting duct have suggested that the defect may be due to a circulating factor that inhibits the action of arginine vasopressin ( AVP ) . ^^^ These studies show that the NDI like defect in Li treatment is small and easily overcome by higher concentrations of AVP and suggests that the concentration defect is at least in part due to increased circulating levels of PTH acting as a partial agonist to AVP and thereby inhibiting its hydroosmotic action . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The mutations of AVP V 2 receptor gene have been clarified in patients with NDI . ^^^ Therefore , both mutations of AVP V 2 receptors and AQP 2 are involved in pathogenesis of NDI . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
We identified three novel mutations of the arginine vasopressin ( AVP ) V 2 receptor ( AVPR 2 ) gene in Japanese families with 10 linked congenital nephrogenic diabetes insipidus ( NDI ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
COS 7 cells were transiently transfected with plasmids encoding mutant forms of the V 2 vasopressin receptors corresponding to mutations [ Y280C , L292P , R337stop , V277A , and G12E ( the latter found in the same kindred with L292P ) ] recently identified in subjects with 10 linked nephrogenic diabetes insipidus ( NDI ) . cAMP response to dDAVP and AVP , saturation binding experiments with [ 3H ] AVP , immunofluorescence , and indirect ELISA studies were performed to characterize the functional consequences of these mutations . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Studies have also shown that the vasopressin V 2 receptor ( V2R ) may modulate AVP mediated vasoconstriction . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
In a large majority of the cases , nephrogenic diabetes insipidus is an 10 linked recessive disorder caused by mutations in the AVP V 2 receptor gene ( AVPR 2 ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Influence of acute elevation of plasma AVP level on rat vasopressin V 2 receptor and aquaporin 2 mRNA expression . ^^^ It is known that vasopressin V 2 receptor ( V2R ) mRNA is downregulated by elevated plasma arginine vasopressin ( AVP ) following chronic osmotic stimulation . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nephrogenic diabetes insipidus ( NDI ) is characterized by resistance of the kidney to the action of arginine vasopressin ( AVP ) ; it may be due to genetic or acquired causes . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
This study demonstrates that AVP increases renal medullary interstitial [ NO ] through vasopressin V 2 receptor stimulation , which in turn elevates blood flow to the renal medulla . . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
All 18 of the NDI associated AVPR 2 alleles tested for function demonstrated diminished response to stimulation with AVP . ^^^ Receptors encoded by eighteen NDI alleles were tested for physiologic signaling activity in response to varying concentrations of arginine vasopressin ( AVP ) in a sensitive cell culture assay . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Oxytocin ( OT ) binds to the vasopressin V 2 receptor ( V2R ) because of its structural similarity to arginine vasopressin ( AVP ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nephrogenic diabetes insipidus ( NDI ) is a rare disease characterized by polyuro polydipsic syndrome ( > 30 ml / kg / day in adult ) related to an inability to concentrate the urine secondary to resistance to the antidiuretic action of vasopressin ( AVP ) or to its V 2 agonist , dDAVP . ^^^ Congenital NDI , familial in most cases , are related in 90 % of cases to mutations of the gene coding for V 2 receptor of AVP ( 10 linked recessive disease ) , and in 10 % of cases , to mutations of the gene encoding for aquaporin 2 ( autosomic recessive disease ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The potentiation by vasopressin was reduced by the antagonist of vasopressin V 1 receptors d ( CH 2 ) 5Tyr ( Me ) AVP ( 10 ( 7 ) M ) , but not by the vasopressin V 2 receptor antagonist d ( CH 2 ) 5D Ile 2 , Ile4AVP ( 10 ( 7 ) M ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nephrogenic diabetes insipidus ( NDI ) is characterized by resistance of the kidneys to the action of arginine vasopressin ( AVP ) ; 10 linked recessive NDI is caused by an inactivating mutation of the vasopressin type 2 ( V 2 ) receptor . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The 10 linked NDI is associated with mutations of the arginine vasopressin receptor type 2 ( AVPR 2 ) gene , which results in resistance to the antidiuretic action of arginine vasopressin ( AVP ) in the renal tubules and collecting ducts . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Intrafamilial phenotype variability in nephrogenic diabetes insipidus . 10 Linked nephrogenic diabetes insipidus ( NDI ) , which accounts for 90 % of inherited cases of NDI , is caused by mutations in the AVPR 2 gene that encodes the arginine vasopressin ( AVP ) receptor type 2 ( V2R ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Polyuria ( 3 , 000 3 , 500 ml / m ( 2 ) per day ) , low urine specific gravity ( 1 . 001 1 . 002 ) , and high plasma arginine vasopressin ( AVP ) ( 18 . 2 pg / ml ) suggested NDI . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nephrogenic Diabetes Insipidus ( NDI ) is characterised by the inability of the kidneys to concentrate urine in response to arginine vasopressin ( AVP ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The identification , characterization , and mutational analysis of three different genes the arginine vasopressin gene ( AVP ) , the arginine vasopressin receptor 2 gene ( AVPR 2 ) , and the vasopressin sensitive water channel gene ( aquaporin 2 [ AQP 2 ] ) provide the basis for understanding of three different hereditary forms of `` pure ' ' diabetes insipidus : Neurohypophyseal diabetes insipidus , 10 linked nephrogenic diabetes insipidus ( NDI ) , and non 10 linked NDI , respectively . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Fluid deprivation testing revealed the presence of AVP resistant NDI . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
In vivo , kidney tissue of rats with lithium induced NDI indeed generated less dDAVP induced cAMP than that of controls , but this could be due to elevated blood AVP levels in rats with lithium induced NDI . ^^^ Indeed , Brattleboro rats , which lack endogenous AVP , with clamped blood dDAVP levels , showed no difference in dDAVP generated cAMP generation between kidneys of rats with lithium induced NDI and control rats . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Most missense AVPR 2 mutations lead to receptors that are trapped intracellularly ; a few mutant receptors reach the cell surface but are unable to bind AVP or to properly trigger an intracellular cyclic adenosine monophosphate signal . ^^^ Nephrogenic diabetes insipidus ( NDI ) , which can be inherited or acquired , is characterized by an inability to concentrate urine despite normal or elevated plasma concentrations of the antidiuretic hormone arginine vasopressin ( AVP ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The disorder nephrogenic diabetes insipidus ( NDI ) is characterized by the kidney ' s inability to concentrate pro urine in response to AVP , which is mostly acquired due to electrolyte disturbances or lithium therapy . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The role of the nonosmotic AVP release in water retention and hypo osmolality / hyponatremia has been demonstrated in patients and experimental animals by administering nonpeptide , orally active vasopressin V 2 receptor antagonists . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
From the present study it may be concluded that , at the doses used , neither c AMP nor its dibutyryl derivative can mimic the action of ADH in NDI as they do in normal subjects . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Ability of these two subjects with NDI to concentrate their urine to Uosm / Posm greater than 1 . 0 in the absence of an increase in urinary cyclic AMP but associated with a decrease in GFR to 50 % normal indicates that urinary concentration was effected by a reduction in GFR rather than a partial response to antidiuretic hormone ( ADH ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
When the adhR mutation expressing the highest ADH and ACDH levels was present together with fadR and atoC mutations ( allowing efficient catabolism of acetoacetyl CoA ) and with an aceX mutation , the resulting strains became capable of using n butanol as sole carbon and energy source . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
These data fail to demonstrate a direct effect of endogenous ADH on renal prostaglandin synthesis in NDI . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
After conducting statistical analyses of the nucleotide sequences of the Adh , Adhr ( Adh related gene ) , and nuclear rRNA genes and a 905 bp segment of mitochondrial DNA , we used Scaptodrosophila as the outgroup . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
In all of the species of the subgroup , a gene of unknown function , Adhr , is located about 300 bp 3 ' to Adh . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Previous work showed that chromosomal breakpoints associated with mutations of the osp locus map to both sides of the alcohol dehydrogenase gene ( Adh ) , suggesting that Adh and the adjacent gene Adhr are nested in osp . ^^^ When hybridized to the osp walk , the 5 ' extension verifies that Adh and Adhr are nested in osp and shows that osp has a transcription unit of > or = 74 kb . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Two tandemly arranged Drosophila genes , alcohol dehydrogenase ( Adh ) and Adh related ( Adhr ) , are transcribed as a dicistronic transcript . ^^^ From transcripts initiated from the Adh promoter , two classes of mRNA are accumulated , one is monocistronic and encodes Adh alone , the other is dicistronic and includes the open reading frames of both Adh and Adhr . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Nonfixed duplication containing the Adh gene and a truncated form of the Adhr gene in the Drosophila funebris species group : different modes of evolution of Adh relative to Adhr in Drosophila . ^^^ The sequence of the genomic region that contains the Adh and Adhr genes of Drosophila funebris was used to demonstrate that both genes are present in species of the funebris group . ^^^ The sequence of this genomic region reveals a 2 . 9 kb tandem duplication which encompasses 1 . 6 kb of the 5 ' flanking region , the entire Adh gene , and two thirds of the first exon of the Adhr gene in D . funebris . ^^^ The codon bias of the Adh gene of D . funebris is among the lowest reported for any Adh gene in the Drosophilidae species and is very similar to that of the Adhr gene . ^^^ The Adhr gene evolves slightly faster than Adh at synonymous positions . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Sequence analysis revealed that Adhr is a member of the Adh multigene family , but does not correspond to any other grapevine Adh described to date . ^^^ These data suggest that Vine 1 insertion in Adhr is specific to 5 . vinifera , and has occurred after the Adh isogene separation , but prior to cultivar development . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Here , we show that in Drosophila lebanonensis , subgenus Scaptodrosophila , Adhr : is also transcribed as a dicistronic transcript with Adh Using degenerate primers designed on the sequence of the known Adhr proteins , we have been able to amplify and sequence a partial sequence of Adhr : in species representative of the whole subgenus Drosophila . ^^^ Adh and Adhr are believed to originate by duplication , and our data suggest that the cotranscription of these two genes was the primitive state , and that their independent transcription in the subgenus Drosophila is derived . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Isolation and characterization of the genomic region from Drosophila kuntzei containing the Adh and Adhr genes . ^^^ The nucleotide sequences of the Adh and Adhr genes of Drosophila kuntzei were derived from combined overlapping sequences of clones isolated from a genomic library and from cloned PCR and inverse PCR fragments . ^^^ Codon bias in Adh and Adhr was comparable and found to be very low compared with other species . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Anterior disk height ratios ( ADHR = ADH / AVH ) and posterior disk height ratios ( PDHR = PDH / PVH ) were calculated from the disk height measurements and were compared to L 4 and L 5 posteroanterior spinal stiffness obtained using a previously validated mechanical impedance stiffness assessment procedure . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
We have employed `` vectorette PCR ' ' to obtain sequence data for orthologous regions of the Alcohol dehydrogenase ( Adh ) , Alcohol dehydrogenase related ( Adhr ) , Glucose dehydrogenase ( Gld ) , and rosy ( ry ) genes ( totaling 7164 bp ) from six melanogaster subgroup species ( D . melanogaster , D . simulans , D . teissieri , D . yakuba , D . erecta , and D . orena ) and three species from subgroups outside the melanogaster species subgroup [ D . eugracilis ( eugracilis subgroup ) , D . mimetica ( suzukii subgroup ) , and D . lutescens ( takahashii subgroup ) ] . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
BACKGROUND : The aim of this study was to describe the urological complications associated with nephrogenic diabetes insipidus ( NDI ) due to a mutation in aquaporin 2 ( AQP 2 ) , a collecting duct protein activated by ADH signalling . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
For example , nephrogenic diabetes insipidus ( NDI ) , the inability to produce concentrated urine , can result from several different malfunctions in the AQP 2 system controlled by anti diuretic hormone ( ADH ) . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
The relationship between third codon position nucleotide content , codon bias , mRNA secondary structure and gene expression in the drosophilid alcohol dehydrogenase genes Adh and Adhr . ^^^ To gain insights into the relationship between codon bias , mRNA secondary structure , third codon position nucleotide distribution , and gene expression , we predicted secondary structures in two related drosophilid genes , Adh and Adhr , which differ in degree of codon bias and level of gene expression . ^^^ In the weakly expressed , weakly biased gene Adhr , the potential for secondary structure formation was found to be much stronger than in the highly expressed , highly biased gene Adh . ^^^ This is consistent with the observation of approximately equal G and C percentages in Adhr ( approximately 31 % across species ) , whereas in Adh the N 3 distribution is shifted toward C ( 42 % across species ) . ^^^ Perturbing the N 3 distribution to approximately equal amounts of A , G , C , and T increases the potential for secondary structure formation in Adh , but decreases it in Adhr . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
Eight of 404 single nucleotide polymorphisms ( SNPs ) in the Adh region were in significant linkage disequilibrium with three ADHR protein alleles . ^^^
Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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Interacting proteins: P01185 and P30518	Pubmed	SVM Score :0.0
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