| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| These include p21ras and its downstream effectors , c raf 1 and MEK , as well as PI3K . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| In this study , we show that oncogenic H Ras requires PI3K and Akt to stimulate the transcriptional activity of NF kappaB . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| H Ras and Rac 1 activate PI3K by targeting the GTPase responsive domain . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| In contrast , H Ras was more effective in activation of phosphatidylinositol 3 kinase ( PI3K ) and AKT . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Wortmannin , an inhibitor of phosphatidylinositol 3 kinase ( PI3K ) , preferentially decreased phosphorylation of externally added Etn in the Ha Ras transformed , but not in the untransformed , fibroblasts . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Coexpression of ( DN ) Rac 1 N 17 and addition of the phosphatidylinositol 3 ' kinase ( PI3K ) inhibitors wortmannin and LY 294002 are in agreement with a tentative model suggesting that , in the signaling pathway from Ha Ras to the cytoskeleton aPKC lambda acts upstream of PI3K and Rac 1 , whereas aPKC zeta functions downstream of PI3K and Rac 1 . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Treatment with LY 294002 , an inhibitor for phosphoinositide 3 OH kinase ( PI3K ) , significantly inhibits Ha Ras ( Val 12 ) induced CD 44 cleavage , whereas that with PD 98059 , an inhibitor for MEK , does not . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| These results indicate that Ras , Bcl 2 , as well as Raf 1 and PI3K pathways play pivotal roles in 5 FU induced apoptosis under Ha ras overexpressed condition . ^^^ Through understanding the mechanism of 5 FU induced apoptosis in tumor cells , a new direction toward the treatment of Ha ras oncogene related cancers with 5 FU at more optimal dosages is possible and combinational therapy with other drugs that suppress PI3K and Bcl 2 activities can also be considered . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Opposite effects of Ha Ras and Ki Ras on radiation induced apoptosis via differential activation of PI3K / Akt and Rac / p38 mitogen activated protein kinase signaling pathways . ^^^ Constitutive activation of phosphoinositide 3 kinase ( PI3K ) and Akt is detected specifically in 12V Ha Ras overexpressing cells . ^^^ The specific PI3K inhibitor LY 294002 inhibits PI3K / Akt signaling and potentiates the radiation induced apoptosis , suggesting that activation of the PI3K / Akt signaling pathway is involved in the increased radio resistance in cells overexpressing 12V Ha Ras . ^^^ Taken together , these findings explain the opposite effects of Ha Ras and Ki Ras on modulation of radiosensitivity , and suggest that differential activation of PI3K / Akt and Rac / p38 MAPK signaling by Ha Ras and Ki Ras may account for the opposing response to the ionizing radiation . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| More recent reports indicate that IL 2R are linked to additional membrane and cytosolic signalling molecules , including glycosylated phosphatidylinositol ( GPI ) , phosphatidylinositol 3 kinase ( PI3K ) , p74c raf and p21ras . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| These phosphorylated substrates directly bind and activate enzymes such as phosphatidylinositol 3 ' kinase ( PI3K ) and the guanine nucleotide exchange factor for p21Ras ( GRB 2 / SOS ) , which are in turn required for insulin stimulated protein synthesis , cell cycle progression , and prevention of apoptosis . ^^^ Yet insulin is still required for the stimulation of general protein synthesis in the presence of constitutively active PKC zeta and in the absence of IRS 1 , suggesting a requirement for the convergence of the IRS 1 / PI3K / PKC zeta pathway with one or more additional pathways emanating from the IR , e . g . , Shc / SOS / p21Ras / mitogen activated protein kinase . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| The inhibitory response of CR 1 in HC 11 cells on beta casein expression after treatment with DIP can be attenuated by B 581 , a peptidomimetic farnesyltransferase inhibitor that blocks p21ras farnesylation and activation , and by the phosphatidylinositol 3 ' kinase ( PI3k ) inhibitor LY 294002 but not by PD 98059 , a MAPK kinase inhibitor that blocks MAPK activation . ^^^ These data suggest that the ability of CR 1 to block lactogenic hormone induced expression of beta casein is mediated through a p21ras dependent , PI3k mediated pathway . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| We have investigated tyrosine kinase mediated activation of phosphatidylinositol 3 kinase ( PI3K ) and compared this with the activation of the p21ras ERK signaling pathway in human eosinophils . ^^^ Finally , using specific inhibitors of both the p21ras ERK and PI3K signaling pathways , a role was demonstrated for PI3K , but not p21ras ERK , in activation of the serum treated zymosan ( STZ ) mediated respiratory burst in IL 5 and PAF primed eosinophils . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| We show that IL 3 mediated activation of FcalphaR ( CD 89 ) requires the activation of PI3K , independent of p21ras activation . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Mitogens act via receptor tyrosine kinase , G protein coupled receptors , or cytokine receptors , to activate p21ras and stimulate two parallel signaling pathways in ASM cells , namely , the extracellular signal regulated kinase ( ERK ) or the phosphatidylinositol 3 kinase ( PI3K ) pathways . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| In this study we demonstrate that in immortalized human thyrocyte cells , adhesion to immobilized fibronectin ( FN ) stimulates DNA synthesis and proliferation through the p21Ras / MAPK pathway , whereas cell survival is mediated by phosphatidylinositol 3 kinase ( PI3K ) signal pathway . ^^^ These results demonstrate that in thyroid cells adhesion to FN regulates proliferation through the p21Ras / MAPK signal pathway , whereas integrin mediated cell survival is mediated by PI3K . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Pure lipopolysaccharide or synthetic lipid A induces activation of p21Ras in primary macrophages through a pathway dependent on Src family kinases and PI3K . ^^^ LPS induced activation of p21Ras was inhibited in the presence of PP 2 , LY 294002 , or wortmannin , suggesting that it depends on the activity of one or more members of the Src kinase family and the subsequent activation of PI3K . ^^^ In that pharmacological inhibitors of PI3K inhibited LPS induced activation of p21Ras , but not activation of ERK , we concluded that LPS induced activation of ERK occurs through a pathway that is not dependent on the activation of p21Ras . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| These results indicate that H Ras activation induces the relocalization and cytoplasmic stabilization of beta catenin by a mechanism involving its interaction with PI3K . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| We found that ROS production by V 12 H Ras expression was mediated by the Ras / phosphatidylinositol 3 kinase ( PI3K ) / Rac1 / NADPH oxidase dependent pathway and that pretreatment of V 12 H Ras transformed cells with an antioxidant ( N acetylcysteine ) and an NADPH oxidase inhibitor ( diphenyleneiodonium ) decreased DNA repair capacity . ^^^ Similarly , treatment with PI3K inhibitors ( wortmannin and LY 294002 ) inhibited the ability of oncogenic H Ras to enhance DNA repair capacity . ^^^ Furthermore , inhibition of the Ras / PI3K / Rac1 / NADPH oxidase pathway resulted in increased sensitivity to cisplatin and UV in V 12 H Ras expressing NIH3T3 cells . ^^^ Taken together , these results provide evidence that oncogenic H Ras activates DNA repair capacity through the Ras / PI3K / Rac1 / NADPH oxidase dependent pathway and that increased ROS production via this signaling pathway is required for enhancement of the DNA repair capacity induced by oncogenic H Ras . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Thus , we propose that H Ras signals followed by Raf 1 / MAPK pathway but not PI3K not only reduces beta ( 1 ) mediated adhesion of osteoblasts to matrix proteins but induces apoptosis presumably via the Fas up regulation and Bcl 2 down regulation . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| In addition , we found that H Ras is also required for the activation of the PI3K / AKT pathway . ^^^ Furthermore , we showed that 5 Crk stimulates the interaction of H Ras with the Ras binding domain in the PI3K p 110 catalytic subunit . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| The human bladder cancer cell line T 24 has amplified and mutated H Ras resulting in sustained PI3K activity and phosphorylation of the downstream target of PI3K , Akt . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| However , downstream receptor tyrosine kinase signaling pathways showed increased amplification rates in resistant tumors for the mitogen activated protein kinase ( FGR / Src 2 , HRAS , and RAF 1 ; P = 0 . 005 ) and phosphatidylinositol 3 ' kinase pathways ( FGR / Src 2 , PI3K , and Akt ; P = 0 . 046 ) . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Pharmacological inhibition studies also revealed that phosphoinositide 3 kinase ( PI3K ) activity is required for H Ras mediated FucT 7 induction . ^^^ However , the ability of H Ras to selectively induce FucT 7 is not a function of the inability of the N or K Ras isoforms to activate Raf or PI3K pathways . ^^^ These studies show that H Ras mediates FucT 7 induction in Jurkat T cells via the activation of the Raf , PI3K , and a distinct , H Ras specific effector signaling pathway . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| In H RAS V 12 cells , radiation caused stronger PI3K / AKT pathway activation compared with that of the ERK1 / 2 pathway , which correlated with H RAS V 12 dependent translocation of PI3K into the plasma membrane . ^^^ Inhibition of PI3K , but not mitogen activated protein kinase / ERK1 / 2 , radiosensitized H RAS V 12 cells . ^^^ Radiation induced activation of the PI3K / AKT pathway in H RAS V 12 cells 2 to 24 hours after exposure was dependent on heregulin stimulated ERBB 3 association with membrane localized PI3K . ^^^ In HCT 116 cells expressing H RAS V 12 , PI3K dependent radioresistance is mediated by both H RAS dependent translocation of PI3K into the plasma membrane and heregulin induced activation of membrane localized PI3K via ERBB3 . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Activated forms of H RAS and K RAS differentially regulate membrane association of PI3K , PDK 1 , and AKT and the effect of therapeutic kinase inhibitors on cell survival . ^^^ Inhibition of H RAS V 12 function , blockade of phosphatidylinositol 3 kinase ( PI3K ) function using small interfering RNA / small molecule inhibitors , or expression of dominant negative AKT abolished radiation induced AKT activation , and radiosensitized these cells . ^^^ In HCT 116 H RAS V 12 cells , PI3K , PDK 1 , and AKT were membrane associated , whereas in parental cells expressing K RAS D 13 , only PDK 1 was membrane bound . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| In order to determine the signaling pathway regulated by HRAS and implicated in the alteration of proliferation of these cells , we used specific chemical inhibitors to inactivate the Raf and PI3K pathways . ^^^ Specific chemical inhibitors for PI3K and MEK activities indicated that both PI3K / AKT and RAF / MEK / ERK pathways are involved in the HRAS oncogene induced reduction of the G 1 phase . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Our results demonstrate that phosphoinositide 3 kinase ( PI3K ) signaling is necessary for oncogenic H Ras mediated reduction of Egr 1 gene expression . ^^^ Our findings suggest a novel signaling mechanism by which prolonged activation of oncogenic H Ras can trigger the loss of tumor suppressor Egr 1 through the PI3K pathway in NIH3T3 fibroblast model cell lines . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Our results show that the activation of a PI3K related enzyme is crucial for H Ras induced MPF activation , whereas the recruitment of either MAPK or RalGDS is not . ^^^ However , although the H Ras / PI3K pathway is functional in Xenopus oocytes , it is not the physiological transducer of progesterone responsible for meiotic resumption . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Both transformation of Rat 1 fibroblasts by 5 Src or K Ras and stable transfection for expression of dominant positive , wild type phosphoinositide 3 kinase ( PI3K ) regulatory subunit p 85 alpha constitutively led to stress fiber disruption , cortical actin recruitment , extensive ruffling , and macropinosome formation , as measured by a selective acceleration of fluid phase endocytosis . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| We have explored this signalling in transformed cells . 5 Src and K Ras activate PI3K and PLC , as demonstrated by in situ production of the corresponding lipid products . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| The present study describes the contributions of the K ras gene mutation and its downstream pathway via phosphatidylinositol 3 OH kinase ( PI3K ) Akt to the cell motility in an immortalized human peripheral airway epithelial cell ( HPL1D ) and lung adenocarcinoma cells ( A 549 , H 820 , TKB 6 , and TKB 14 ) . ^^^ These results suggest that the K ras gene mutation could enhance the motility of neoplastic cells through a pathway involving PI3K Akt . ^^^ Actually , among the surgically resected lung tumors , the adenocarcinomas with the K ras gene mutation tended to show a higher frequency and intensity of immunoreactivity for phosphorylated Akt ( p ser473Akt ) than those without the mutation , supporting the in vitro observation that the mutated K ras can activate the PI3K Akt pathway . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Here , we elucidated the roles of PI3K and mTOR in K Ras mediated transformation of IECs ( IEC 6 ) . ^^^ Induction of K Ras activated PI3K and mTOR in IECs . p 70 ribosomal protein S 6 kinase activity was induced by K Ras in a PI3K and mTOR dependent manner . ^^^ Thus , our results suggest that K Ras mediated transformation of IECs involves activation of the PI3K / mTOR pathway . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Inhibition of phosphoinositide 3 OH kinase ( PI3K ) abolished K Ras actions on ENaC . ^^^ Activation of ENaC by K Ras , moreover , was sensitive to co expression of dominant negative p 85 ( PI3K ) . ^^^ Constitutively active PI3K activated ENaC independent of K Ras with the effects of PI3K and K Ras on ENaC not being additive . ^^^ We conclude that K Ras activates ENaC via the PI3K cascade . . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| We now report that in human colorectal cancer cells , TF expression is under control of 2 major transforming events driving disease progression ( activation of K ras oncogene and inactivation of the p 53 tumor suppressor ) , in a manner dependent on MEK / mitogen activated protein kinase ( MAPK ) and phosphatidylinositol 3 ' kinase ( PI3K ) . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Knocking down PTEN expression in the HEC 1B cell line , which possesses both K Ras and PIK3CA mutations , further enhances phosphorylation of Akt ( Ser 473 ) , indicating that double mutation of PIK3CA and PTEN has an additive effect on PI3K activation . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| Activation of the N Ras PI3K Akt mTOR pathway by hepatitis C virus : control of cell survival and viral replication . ^^^ N Ras is an activator of the phosphatidylinositol 3 kinase ( PI3K ) Akt pathway . ^^^ Taken together , these data suggest that increased N Ras levels in subcellular sites of HCV replication and stimulation of the prosurvival PI3K Akt pathway and mTOR by HCV not only protect cells against apoptosis but also contribute to the maintenance of steady state levels of HCV replication . ^^^ |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P01112 and P27986 |
Pubmed |
SVM Score :0.0 |
| NA |
|