| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| All CB units were collected from the umbilical cord vein immediately after vaginal full term delivery or caesarean section , then frozen and stored in liquid nitrogen . 1940 CB units were stored unseparated , the other 1296 were volume reduced using Hetastarch ( HES ) with a mean recovery of 85 + / 13 % of the nucleated cells , 86 + / 12 % and 84 + / 13 % for CFC and CD34+ cells , respectively . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Here , we report that PTPN 11 lesions occur in childhood acute lymphoblastic leukemia ( ALL ) . ^^^ PTPN 11 , NRAS , and KRAS 2 mutations were largely mutually exclusive and accounted for one third of common ALL cases . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| She provides an overview of leukemias that are common in pediatric malignancies but rarely observed in adults , including the TEL AML 1 ( ETV 6 RUNX1 ) fusion associated with pediatric B cell ALL , the OTT MAL fusion associated with infant megakaryoblastic leukemia , PTPN 11 mutations in juvenile myelomonocytic leukemia , and MLL fusion genes in leukemogenesis , among others . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Recently , PTPN 11 mutations have been reported in children with acute lymphoblastic leukemia ( ALL ) . ^^^ In the present study , we investigated the prevalence of mutations in PTPN 11 , RAS and FLT 3 in samples from 95 Japanese children with ALL . ^^^ We observed exon 3 and 8 missense mutations of PTPN 11 in 6 children with B precursor ALL . ^^^ One patient with Down syndrome and ALL had PTPN 11 mutation . ^^^ These data suggest that PTPN 11 mutation may play an important role for leukemogenesis in a proportion of children with ALL , particularly B precursor ALL . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Based on these data we assume that SHIP and SHP 2 recruited in vivo to the Fc gamma RIIb co clustered BCR are responsible for the Fc gamma RIIb mediated negative regulation of human B cell activation . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| B cell antigen receptor ( BCR ) mediated formation of a SHP 2 pp 120 complex and its inhibition by Fc gamma RIIB 1 BCR coligation . ^^^ However , coligation of Fc gamma RIIB 1 with B cell Ag receptors ( BCR ) inhibits BCR mediated signaling by a mechanism that may involve recruitment of phosphatases SHP 1 , SHP 2 , and the SH 2 containing inositol 5 ' phosphatase ( SHIP ) to the phosphorylated Fc gamma RIIB 1 immunoreceptor tyrosine based inhibitory motif . ^^^ The role of SHP 2 in BCR mediated cell activation and in Fc gamma RIIB 1 mediated inhibitory signaling is unclear . ^^^ BCR stimulation induced the association of SHP 2 with a single major tyrosyl phosphorylated molecule ( pp 120 ) that had an apparent molecular mass of 120 kDa . ^^^ Coligation of Fc gamma RIIB 1 with BCR led to a rapid decrease in SHP 2 association with pp 120 . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| From the lysates of BL 41 cells SHP 2 and phosphatidylinositol 3 kinase ( PI 3 K ) , as well as the protein tyrosine kinase ( PTK ) Lyn bind both to the BCR co clustered Fc gamma RIIb and to its P ITIM peptide . ^^^ The results indicate that SHP 2 bound to and activated by the BCR co clustered Fc gamma RIIb , may down regulate PI 3 K activity by dephosphorylating a yet unidentified regulatory molecule , which recruits PI 3 K to the cell membrane . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Gab 1 expression also increased BCR induced tyrosine phosphorylation of SHP 2 as well as the binding of Grb 2 to SHP 2 . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| SHP 2 , activated upon the binding to FcgammaRIIb SHIP complex , partially dephosphorylates Gab 1 , resulting in the release of PtdIns 3 K and ultimately in the inhibition of downstream activation pathways in BCR / FcgammaRIIb co aggregated cells . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| This tyrosine was phosphorylated and recruited src homology 2 domain containing tyrosine phosphatase 2 ( SHP 2 ) on coligation of PD 1 with BCR . ^^^ These results show that PD 1 can inhibit BCR signaling by recruiting SHP 2 to its phosphotyrosine and dephosphorylating key signal transducers of BCR signaling . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| We demonstrate that SHIP is necessary for human FcgammaRIIB to inhibit BCR signaling , and can not be replaced by SHP 1 or SHP 2 . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| In untreated CML patients ( at diagnosis or in relapse ) we find about the same number of CFC per 1 . 10 ( 6 ) nucleated cells in the blood and bone marrow ( sometimes , slightly greater in the blood than in the marrow ) . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Cellular recovery , maturation , and colony forming cell ( CFC ) generation patterns of bone marrow cells from 23 patients with acute , subacute , and chronic myeloid leukemia ( AML , SML , and CML ) were studied using liquid and agar culture techniques . ^^^ The normally observed increase in CFC during liquid culture was absent in most leukemic marrow samples studied ( 3 of 4 AML , 4 of 6 SML , and 2 of 3 CML ) . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Bone marrow CFC concentration tended to be increased in chronic myeloid leukaemia ( CML ) and polycythaemia vera ( PV ) , but decreased in myelofibrosis , erythroleukaemia , paroxysmal nocturnal haemoglobinuria ( PNH ) and the aplastic phase of aplastic anaemia . ^^^ The proportion of CFC in DNA synthesis was decreased in CML , myelofibrosis and aplastic anaemia , but increased in blastic transformation , PV , PNH and during regeneration from aplastic anaemia . ^^^ The proportion of CFC in DNA synthesis in bone marrow from patients with CML in blastic transformation was directly related to the percentage of myeloblasts in the bone marrow . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Six patients with chronic myelogenous leukaemia ( CML ) were treated by repeated continuous flow centrifuge ( CFC ) leucapheresis . ^^^ Balancing potential benefits against definite costs to the patient , CFC leucapheresis seems justified in only a minority of CML patients . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Cultures in agar in the study of chronic myeloid leukemia ( CML ) : analysis of splenic and medullary colony forming cells ( CFC ) ] . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Colony forming cells ( CFC ) and CFC in S phase were assayed in chronic myeloid leukemia ( CML ) . ^^^ A correlation was found between leukocyte counts and CFC of blood , suggesting that the leukocytosis of CML depended on expansion of the committed granulopoietic stem cell compartment . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Therefore , we tested whether these leukemias could be distinguished with respect to their involvement of immature precursors by studying colony forming cells ( CFC ) and their precursors from four glucose 6 phosphate dehydrogenase ( G6PD ) heterozygous patients with AML and five patients with CML . ^^^ In contrast , in four of five patients with CML , cultures of both the CD 33 CD34+ and CD33+CD34+ populations produced CFC in LTC that were almost entirely clonal in origin , whereas in the fifth patient a substantial number originated from nonclonal stem cells . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| In only one case of CML an agar culture from peripheral blood cells led to the presence of circulating colony forming cells ( CFC ) capable of generating monocytic and eosinophilic CAN in cultures . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Leukapheresis by continuous flow centrifugation ( CFC ) in patients with chronic myelocytic leukemia ( CML ) . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| A monoclonal antibody , anti N 901 , was produced by fusing NS 1 myeloma cells with spleen cells of a mouse immunized with human CML cells . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| CFC derived from bone marrow and peripheral blood of normal subjects and patients with chronic myeloid leukemia ( CML ) were studied . ^^^ Sensitivities to m AMSA did not differ significantly between normal marrow and blood CFC , between normal and CML CFC , or between CML CFC obtained from patients with leukemias in chronic phase and blast transformation . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| We demonstrate that treatment with interferon alpha induces a significant , dose dependent increase in the adhesion of primitive long term culture initiating cells and committed colony forming cells ( CFC ) from CML bone marrow to normal stroma . ^^^ Adhesion of CFC seen after interferon alpha treatment could be inhibited by blocking antibodies directed at the alpha 4 , alpha 5 , and beta 1 integrins and vascular cell adhesion molecule , but not CD 44 or intracellular adhesion molecule , suggesting that interferon alpha induces normalization of progenitor stroma interactions in CML . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| We show here that , in contrast to normal colony forming cells ( CFC ) , the percentage of malignant CML CFC in S phase was not significantly reduced following coculture with stromal layers . ^^^ However , IFN alpha treatment resulted in a significant reduction in the proliferation of CML CFC on coculture with stroma . ^^^ This effect was not because of a direct antiproliferative effect of IFN alpha on CML CFC because the proliferation of IFN alpha treated CML CFC kept in suspension culture was not reduced . ^^^ In the first set of experiments , we demonstrated that proliferation of IFN alpha treated CML CFC , but not untreated CML CFC , was significantly reduced following coculture with 33 / 66 kD and 75 kD FN fragments , recognized by alpha 4 beta 1 and alpha 5 beta 1 integrins respectively . ^^^ In a second set of experiments , we demonstrate that direct stimulation of integrin receptors by crosslinking with blocking antibodies to alpha 4 , alpha 5 , and beta 1 integrins and secondary goat antimouse antibodies resulted in significant reduction in proliferation of normal and IFN alpha treated CML progenitors but not untreated CML CFC . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| In contrast , ANK from eight patients with CML with potent cytotoxicity against NK sensitive ( K 562 ) NK resistant ( Raji ) tumor targets exhibited an ANK dose dependent suppression of both CFC and LTCIC . ^^^ Interestingly , ANK from two patients with CML who exhibited diminished cytotoxicity also did not suppress autologous CFC and LTCIC . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Elevated numbers of primitive Philadelphia chromosome positive ( Ph+ ) progenitors , including long term culture initiating cells ( LTC IC ) as well as colony forming cells ( CFC ) , have been previously described in the blood of patients with chronic myeloid leukemia ( CML ) in chronic phase with high white blood cell counts . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Incubation of CML marrow CD34+HLA DR+ cells with Tyrphostin AG 957 at concentrations that did not affect colony forming cells ( CFC ) viability , but which partly inhibited p210BCR / ABL kinase activity , significantly increased CML CFC adhesion to stroma and alpha4beta1 and alpha5beta1 integrin binding fragments of fibronectin ( FN ) . ^^^ CML CFC proliferation , unlike that of normal CFC , is not inhibited following integrin receptor engagement with FN or anti integrin antibodies . ^^^ AG 957 did not alter CML CFC proliferation by itself , but resulted in significant inhibition of CML CFC proliferation following integrin engagement . ^^^ Another PTK inhibitor , Tyrphostin AG 555 , which does not have anti p210BCR / ABL kinase activity , did not affect CML CFC adhesion or proliferation . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Bone marrow and peripheral blood cells of eight newly diagnosed CML patients were analysed for clonogenic colony forming cells ( CFC ) and very early progenitor cells , i . e . long term culture initiating cells ( LTC IC ) . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| In contrast to normal CFC , chronic myelogenous leukemia ( CML ) Ph+ CFC adhere significantly less to either stroma or FN . ^^^ CML Ph+ CFC proliferation is also not inhibited by coculture with stroma or FN . ^^^ We have previously demonstrated that beta 1 dependent adhesion to and subsequent proliferation inhibition by FN can be restored when CML Ph+ CFC are incubated with the beta 1 integrin activating antibody , 8A2 , and demonstrated a role for the alpha5beta1 integrin in this phenomenon . ^^^ Since the integrin alpha4beta1 and the proteoglycan form of CD 44 may cooperate in establishing normal CFC adhesion to FN , we examined if treatment of CML Ph+ CFC with 8A2 also restores the cooperativity between beta 1 integrins and CD 44 . ^^^ We demonstrate that 8A2 induces adhesion of CML Ph+ CFC not only to intact FN but also to alpha4beta1 , alpha5beta1 , and proteoglycan binding fragments of FN . 8A2 induced adhesion to these fragments and peptides also results in a significant inhibition of the proliferation of CML Ph+ CFC . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| In contrast to the LTC IC , CFC were consistently amplified in cultures initiated with CML derived CD34+CD38 cells and the additional CFC present after 10 days were , like the starting population of CFC , almost exclusively Ph+ regardless of the genotype ( s ) of the LTC IC in the original CML samples . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| The human CFC and total human cells present in the BM of the NOD / SCID mice transplanted with CML cells also contained higher proportions of leukemic cells than were obtained in the SCID model , and NOD / SCID mice could be repopulated with transplants of enriched CD34+ cells from patients with CML . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| CFC from PBMNC of the same lineage commitment were simultaneously present in plastic adherent and nonadherent cell fractions , indicating that their surface charges might be different and , on the other hand , that different lineage commitment precursors can be present in either of the fractions irrespective of CML or blood origin . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| The clonogenic cells of chronic myeloid leukaemia ( CML ) , unlike normal haemopoietic colony forming cells ( CFC ) , are resistant to the growth inhibitory effects of the chemokine , macrophage inflammatory protein 1alpha ( MIP 1alpha ) . ^^^ Here , we tested the hypothesis that MIP 1alpha protects normal , but not CML , CFC from the cytotoxic effects of the cell cycle active drug cytosine arabinoside ( Ara C ) . ^^^ Using a 24 h Ara C protection assay we showed that MIP 1alpha confers protection to normal CFC but also sensitizes CML CFC to Ara C . ^^^ Our data suggest that the unresponsiveness of CML CFC to the growth inhibitory effect of MIP 1alpha is not caused by a lack of MIP 1alpha receptor or total uncoupling of the MIP 1alpha responsiveness but may be due to an intracellular signalling defect downstream of the receptors . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Finally , similar to what was seen for K 562 cells , transduction of CML CFC improved two to three fold with either CH 296 or lipids , whereas the combination had synergistic effects . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Short term coculture ( 5 days ) of CML or normal monocytes with CML or normal peripheral blood mononuclear cells ( PBMNC ) / CD34+ cells as targets resulted in a significant inhibition of colony forming cell ( CFC ) growth . ^^^ Coculture conditioned medium ( CCM ) from 5 days cocultures of normal or CML CD14+ monocytes with CD34+ cells were likewise inhibitory to CFC . ^^^ In 5 week long term cocultures of monocytes in direct contact with normal bone marrow ( BM ) progenitors , CML monocytes reduced the proportion of long term cultured CFC ( LTC CFC ) significantly to 52 % of the controls , while normal monocytes had a less pronounced inhibitory effect ( 89 % of the controls ) on LTC CFC . ^^^ Reduction of LTC CFC was great when CML monocytes and target cells were separated by a transwell membrane as compared to control cultures in the absence of CD14+ cells ( 53 . 5 vs . 9 % ) . ^^^ CCM from 5 week cocultures of normal or CML CD14+ monocytes with CD34+ progenitors from bone marrow ( BM ) cells were also inhibitory to CFC . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| These data provide evidence for a differential effect of IFN alpha on the growth of CML and normal CFC cells in a stromal context and suggest that an alteration in the response of CML progenitor cells to FL is important in the explanation of this differential effect . . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| The excessive production of leukemic CFC that is seen in most CML patients at diagnosis may be explained at least in part by their increased proliferative activity . ^^^ Although the molecular basis of these changes in CML progenitor regulation is not fully understood at the level of the primitive CFC compartment , a selective inability of CML progenitors to be inhibited by certain C C type chemokines has been demonstrated . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Culture of CML and normal CD 34 ( + ) cells with different GF for 7 days resulted in similar expansion of committed progenitors ( CFC ) . ^^^ CML CD 34 ( + ) cells enter into cycle more quickly than normal CD 34 ( + ) cells and CML CFC expansion was accelerated compared to normal CFC . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| CML colony forming cells ( CFC ) demonstrated increased migration across FN coated transwells compared with normal CFC in the absence of GF . ^^^ The addition of stem cell factor resulted in enhanced migration of CML and normal CFC on FN . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| After 8 days of culture , their proliferation , capacity for colony forming cell ( CFC ) generation in secondary cultures ( pre CFC ) , and phenotype ( CD 34 and platelet activating factor receptor [ PAF R ] ) were compared with those of normal cells , and tyrosine phosphorylation in CML cells was measured . ^^^ Hypoxia inhibits the proliferation of CD 34 ( + ) cells and preserves the pre CFC capacity and cell surface CD 34 expression of CML cells better than normoxia . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| In spite of this latter observation , no significant differences were observed in terms of the absolute number of CFC when comparing Populations 1 and 2 from CML patients and normal subjects . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Laboratory correlative studies were performed prior to treatment and at intervals during treatment to evaluate effects on maturation and differentiation , and on CML progenitor cell growth by assessment of colony forming cells ( CFC ) . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| Enhanced sensitivity to inhibition of SHP 2 , STAT 5 , and Gab 2 expression in chronic myeloid leukemia ( CML ) . ^^^ Similarly , colony formation of purified primary CML but not of normal CD34+ colony forming cells is specifically reduced by inhibition of SHP 2 , STAT 5 , and Gab 2 expression , respectively . ^^^ |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P11274 and Q06124 |
Pubmed |
SVM Score :0.0 |
| NA |
|