Pubmed abstracts for Protein-Protein Interaction search result :

Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Several sequences homologous to the c fos RB control element were identified in the TGF beta 1 promoter . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
However , the stimulatory effects of CamKs and Rb on c fos are blocked by overexpression of both proteins . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
A 30 base pair element within the c fos promoter , termed the RCE ( retinoblastoma control element ) , has previously been shown to be the target of transcriptional regulation by the product of the retinoblastoma ( Rb ) gene . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Here we co inject RB protein with c myc , EJ ras , c fos or c jun protein . ^^^ Co injection of c myc , but not EJ ras , c fos or c jun , inhibits the ability of Rb to arrest the cell cycle . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Thus , taken with previous results , then amongst the nuclear oncogenes or tumor suppressor genes , c myc , RB , c fos , and c myb , only c myc and RB expression exhibit early regulation during induced HL 60 cell differentiation . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Two copies of a motif ( the retinoblastoma control element ) similar to that found in the human c fos , transforming growth factor beta 1 , and c myc promoters are responsible for conferring Rb regulation to the fourth promoter of the insulinlike growth factor 2 gene . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
In similar transient transfection experiments in HeLa cells , overexpression of the wt human retinoblastoma susceptibility gene product , RB , was found to repress the serum induced IL 6 ( 225 to +13 ) , c fos ( 711 to +42 ) , and beta actin ( 3400 to +912 ) promoters but not the PRV induced IL 6 ( 110 to +13 ) or the serum induced MHC ( 528 to 38 ) promoters . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The c fos expression , which is presumed to be regulated negatively by RB , was not stimulated in partially senescent TIG 1 cells by treatment with the antisense Rb oligomer . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The promoter of the c fos gene might be a target for regulation by Rb , because both 5 fos and RB 1 are associated with the induction of osteosarcomas in mice and humans , respectively . ^^^ Here we report that Rb can repress c fos expression and AP 1 transcriptional activity in both serum induced and cycling 3T3 cells . ^^^ We have mapped a cis acting element in the human c fos promoter that can confer repression by Rb to a heterologous promoter . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
We performed experiments to determine the effects of ionizing radiation exposure on expression of genes such as beta actin , c fos , histone H 4 , c myc , c jun , Rb , and p 53 after exposure of Syrian hamster embryo ( SHE ) cells to the protein synthesis inhibitor cycloheximide . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The results of slot blot hybridizations showed that tumor promoter TPA induced c fos and c myc expressions in mouse fibroblast cell line BALB / 3T3 and rat liver , decreased the levels of Rb RNA in BALB / 3T3 cell line and of alpha 1 I 3 RNA in rat liver . ^^^ Tumor promoters induced the expressions of c fos and c myc which were positively related to cancer formation and inhibited the expressions of Rb and alpha 1 I 3 which were negatively related to cancer formation . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Cells en route to apoptosis are characterized by the upregulation of c fos , c myc , c jun , cdc 2 , and RB phosphorylation , resembling events of early cell cycle traverse . ^^^ Investigating the regulation of early cell cycle genes during this process , we found that c myc , c jun , c fos , and cdc 2 protein presence is induced after serum deprivation , when the phosphorylated form of the RB protein also appears . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
AG 879 also inhibited NGF induced PLC gamma 1 phosphorylation , phosphatidylinositol 3 ( PI 3 ) kinase activation , the association of the tyrosine phosphorylated proteins pp 100 and pp 110 with the p 85 subunit of PI 3 kinase , mitogen activated protein and raf 1 kinases , and c fos induction . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The percentage of myeloma patients with a greater than normal incidence of plasma cells expressing these proteins was 53 % for c myc , 28 % for Rb , 28 % for bcl 2 , 27 % for c fos , 24 % for p 53 wild , 22 % for p 53 mutant , 13 % for c neu and 13 % for pan ras . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The transient expression of the retinoblastoma protein ( Rb ) regulates the transcription of a variety of growth control genes , including c fos , c myc , and the gene for transforming growth factor beta 1 via discrete promoter sequences termed retinoblastoma control elements ( RCE ) . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Overexpression of c fos induces expression of the retinoblastoma tumor suppressor gene Rb in transfected cells . ^^^ We investigated whether Rb expression was dependent on the expression level of the immediate early cell growth gene , c fos , which is transiently expressed as cells re enter G 1 phase from quiescence . ^^^ To explore the functional relationship between c fos and Rb , a eukaryotic expression plasmid was constructed containing the c fos gene under control of the SV 40 promoter complex . ^^^ Because one possible function of RB may be to act as a brake on cell growth , it is possible that overexpression of RB acts as an inhibitory counter activity to overexpression of the growth promoting activity of c fos . ^^^ This possible balancing activity of Rb was further suggested when Rb protein expression levels were measured in different clonal lines of RSfos transfected cells overexpressing increasing levels of c fos . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Yet a normal function of EGF receptors was evidenced , since the immediate early gene c fos was rapidly induced , suggesting a constitutive expression of rhoB in these cell lines bypassing the regulation by EGF . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
We further demonstrated that gip 2 stimulates the negative silencer activity of the retinoblastoma ( Rb ) control element ( RCE ) and inhibits the c fos promoter activity through RCE located in the c fos promoter region . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
AGM 1470 does not inhibit early G 1 mitogenic events , such as cellular protein tyrosyl phosphorylation or the expression of immediate early genes c fos and c myc , but potently inhibits phosphorylation of RB protein , a tumor suppressor retinoblastoma gene product . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
We also found that apoptotic mouse 3T3 fibroblasts express c fos , c myc , c jun , and cdc 2 , as well as the upregulation of RB phosphorylation and BrdU incorporation , just before final DNA fragmentation and death . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The induced differentiation occurs with down regulation of c fos protein and an accompanying up regulation of RB protein expression , consistent with a possible need for up regulated RB expression to maintain a given differentiated phenotype and suppress transcriptional activators that might typically be associated with proliferation . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The retinoblastoma ( RB ) protein is implicated in transcriptional regulation of at least five cellular genes , including c fos , c myc , and transforming growth factor beta 1 . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Other less clearly defined abnormalities in astrocytomas include mutations of the retinoblastoma ( RB ) gene and overexpression of platelet derived growth factor ; transforming growth factor alpha and beta ; the c erb B 1 , c myc , ras , c fos , and ros oncogenes ; and insulin like growth factor 1 and 2 . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Twenty four tumor tissues and one human cell strain established from an osteosarcoma were examined by Southern blot analysis using a recessive oncogene ( p0 . 9R and p3 . 8R derived from a cDNA of the Rb gene ) and eight dominant oncogenes ( c myc , c K ras , c fos , c raf 1 , c fms , c sis , N myc , and c erb B ) as probes . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The putative transregulatory activity of the RB ( retinoblastoma tumor suppressor ) gene product on the expression of the c myc and c fos proteins during the cell cycle was assessed in HL 60 promyelocytic leukemia cells . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Other downstream targets for ceramide action include Cox , IL 6 and IL 2 gene expression , PKC zeta , Vav , Rb , c Myc , c Fos , c Jun and other transcriptional regulators . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Nine of these genes ( H ras , c myc , c fos , c jun , junB , N myc , c abl , c yes , and p 53 ) were expressed in epithelial cells , whereas two ( RB 1 and N ras ) were not . expression of four other genes ( c src , K ras , c raf , and c myb ) was observed , but the intensity of these bands was too low for densitometric analysis . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The c fos promoter is negatively regulated by the retinoblastoma ( Rb ) susceptibility gene encoded protein as well as by other genes involved in the control of transcription , cell cycle regulation and neoplastic transformation . ^^^ We have examined by immunohistochemistry the c Fos and c Jun proteins in five cases of retinoblastoma in order to evaluate eventual alterations in their expression in vivo , possibly related to a gene mutation or to loss of Rb negative control . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Our results suggest that alterations of c myc , N myc , and c fos oncogenes occur in osteosarcomas , in addition to those previously described for the tumor suppressor genes RB and p 53 . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
We and others have previously shown that the SP family of mammalian transcription factors regulates the transcription of a number of genes whose activities are governed by the product of the retinoblastoma ( Rb ) susceptibility gene , including c FOS , c MYC , TGFbeta 1 , IGF 2 , and c JUN . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Immunoblot analyses were performed with specific antibody against BCL 2 , BCL XS / L , BAX , JUNB , c JUN , ICH 1L , c FOS , RB , CDK 2 , and p 53 . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Transcription factor , SP 1 , and retinoblastoma susceptible gene product , RB , are substrates of SPase while other nuclear factors such as c Jun and c Fos are not . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The normal range of expression for each of 6 oncoproteins ( c myc , c fos , c neu , bcl 2 , p ras , p 53 mutant ) and 2 tumour suppressor gene products ( p 53 wild and Rb ) was determined in plasma cells from 33 normal bone marrows . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
These antigens included the labelling index ( LI , bromodeoxyuridine ) , number of nucleoside transporter per cell , p glycoprotein ( JSB 1 ) , and oncoprotein expression ( c myc , c fos , c neu , bcl 2 , p ras , p53m , p 53w , and Rb ) . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
But the transcriptional expression of c fos , c myc , c jun , H ras , 5 erb B oncogenes and Rb antioncogene in atherosclerotic lesion has not yet been reported . ^^^ The results showed that the atherosclerotic plaques contained 3 4 fold more 5 sis , c fos and c myc mRNA ( P < 0 . 01 ) , 2 fold more c jun and H ras mRNA ( P < 0 . 01 ) , and less Rb mRNA ( P < 0 . 05 ) than those in the normal aortic arteries . ^^^ These results indicate that the abnormal expression of 5 sis , c myc , c fos , c jun , and H ras oncogenes and Rb antioncogene may play an important role in arterial SMC proliferation and pathogenesis of atherosclerosis . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Interaction of the Rb tumor suppressor protein with the c fos promoter in c fos transfected cells overexpressing c fos and Rb . ^^^ The Rb tumor suppressor protein is overexpressed in HeLa cell lines permanently transfected with a constitutively expressed c fos gene . ^^^ CP 17 14 cell overexpression of Rb may be due to a balancing response to overexpression of the stimulatory effects of c fos overexpression on transcription . ^^^ The cis acting retinoblastoma control element ( RCE , 97 to 86 bp ) in the human c fos promoter is thought to allow regulation of c fos by Rb . ^^^ These results suggested the presence of Rb and / or Rb like peptides involved in complex formation and the presence of multiple variants of RCE binding complexes in response to c fos over expression . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Sodium butyrate ( SB ) is a potent biological modifier that can induce diverse effects including growth inhibition , differentiation , or apoptosis of many cell types including retinoblastoma ( Rb ) , and modulation of genes such as c fos and p 53 . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Therefore , we studied protein expression of 6 cell cycle regulatory proteins ( Rb , p 16 , p 21 , p 27 , cyclin D 1 , and cyclin E ) in protein extracts from 53 breast cancer samples and 4 mammary cell lines and correlated the data with expression of the 7 AP 1 family members ( c jun , junB , junD , c fos , fosB , fra 1 , and fra 2 ) as determined in a previous study . ^^^ After Western blot analysis , we found significant associations between members of both groups : whereas c jun was associated with Rb expression ( p = 0 . 002 ) , strong junD and c fos expression correlated with high cyclin E reactivity ( p = 0 . 017 and p = 0 . 013 , respectively ) . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Insulin induced clonal expansion , Rb phosphorylation and adipocyte differentiation were all inhibited by the PI 3 kinase inhibitors and rapamycin , but not the MEK inhibitor , whereas the MEK inhibitor , but not PI 3 kinase inhibitors or rapamycin , decreased c fos induction . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
RB , CDK 4 , MDM 2 , c myc , c fos , and p 53 gene expression , which are the major genes involved in osteosarcoma susceptibility , were studied . ^^^ Our results revealed an inactive form of p 53 sporadically seen in the samples , a total loss of Rb protein expression , an increased expression of Cdk 4 , MDM 2 , c fos , and c myc proteins which literature currently reports being the principal alterations found in osteosarcoma . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Despite this oncogenic lesion , Y 1 cells retain tight regulatory mechanisms of cell cycle control typified by the sequential events comprising the mitogenic response triggered by FGF 2 in G0 / G1 arrested Y 1 cells : 1 ) activation of ERK1 / 2 and PI3K , by 5 minutes ; 2 ) induction of c Fos and c Myc proteins by 2 hours ; 3 ) induction of cyclin D 1 protein by 5 hours ; 4 ) phosphorylation of Rb protein between 6 and 8 hours ; 5 ) onset of DNA synthesis by 8 9 hours . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Overexpression of c fos and Rb proteins in radiation induced skin ulcers . ^^^ Immunohistochemical studies were performed on 72 radiation induced skin ulcer specimens using c fos and Rb protein polyclonal antibodies . ^^^ We found that the overexpression rate of c fos was 45 . 8 % and of Rb was 63 . 9 % . ^^^ Our results suggest that the changes in c fos and Rb proteins may be related to the poor healing of radiation induced skin ulcers . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
In general , the checkpoint for replicative senescence is found at the G ( 1 ) / S border , controlled by the modulation of a battery of proteins , typified by gaining inhibitors of cell cycle traverse , such as cyclin dependent kinases or RB hyperphosphorylation , and losing pro proliferation gene expressions such as c fos , c myc , and a cadre of proliferation dependent kinases . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Immunoblotting analyses demonstrated that Epimedin C caused a decreased expression of hyperphosphorylated retinoblastoma ( Rb ) protein , cyclin D 1 , c Myc , and c Fos . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The possibility that local diffusible factors play a major role in determining tissue specific effects of estrogen was examined by transplanting uterine tissues into the brain , muscle and kidney of adult rats and then comparing the effects of estrogen on the incorporation of [ 3H ] thymidine and the expression of Fos , cdc 2 and Rb like immunoreactivity ( IR ) on native and transplanted uterine tissues , as well as in estrogen concentrating regions of the brain adjacent to the uterine grafts . ^^^ In native uteri , estrogen treatment stimulated Fos , cdc 2 , and Rb like IR , as well as [ 3H ] thymidine incorporation , within lumenal and glandular epithelial cells . ^^^ When uterine tissues were transplanted to brain , Fos , cdc 2 , and Rb like IR epithelial cells , as well as many [ 3H ] thymidine incorporating uterine epithelial cells , were observed in all estrogen treated animals and in some non estrogen treated animals as well . ^^^ In contrast , estrogen did not stimulate cell division and did not induce Fos , cdc 2 , or Rb like IR within estrogen concentrating neuronal regions of the ventromedial hypothalamus . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
We examined the expression of the nuclear `` early response ' ' genes jun and fos and the `` tumor suppressor ' ' retinoblastoma ( Rb ) gene , as well as genes involved in invasion ( major excreted protein [ MEP ] , tissue inhibitor of metalloproteinases [ TIMP ] ) , and cell adhesion ( secreted phosphoprotein 1 [ SPP 1 ; also known as osteopontin ] ) . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
A summary of what is known about some of these regulatory oncogenes ( fos , jun , myc , and Rb 1 ) and where they might function in the progression of a cell from G 0 to G 1 and G 1 to S is presented . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The delayed G 0 > S progression in fos expressing cells was accompanied by the inappropriate levels or kinetics of expression of several cell cycle regulated genes ( cyclin D 1 , cdc 2 , cdk 2 , cdk 4 and rb ) . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The comparison between parental , revertant and back revertant cells has revealed that , beyond the mutation in codon 61 of N ras , two groups of genes appear to be also implicated in the transformation process of XP 44 RO ( Mel ) cells : one group , comprising pim A , trk , Rb and p 53 , whose expression is independent of the cell selection conditions ; the other group , comprising Ha ras , N ras , neu 1 , fos and met H , whose expression is more or less dependent upon such conditions . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Recent experimental results suggest that RB 1 may exert its growth suppressing activity by regulating the transcription of a variety of growth related genes , including FOS , MYC , and TGFBI . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Transcripts coding for transcription factors ( RB , P 53 , FOS , MYC , MYB , ERBA , REL ) , growth factors ( FGF 1 , FGF 2 , INT 2 , TGFA , TGFB , PDGF , IGF 1 , IGF 2 ) , interleukins , ( IL 1 , IL 2 , IL 3 , IL 4 , IL 6 , TNF ) , growth factor receptors or cytosolic protein kinases ( RAF , PIM , FES , MET , SRC , ROS , TRK , KIT , CSFR , IGFR , PDGFR , EGFR , NEU ) were quantified in cultured human mammary fibroblasts from normal tissues , benign tumours , carcinomas and post radiation fibrosis lesions by slot blot autoradiography and image analysis . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
The present report summarizes the current molecular biological aspects of several oncogenes ( erbB , ras , myc , raf , fos , jun , bcl , mdm 2 , myb , kit CSF1R , met ) and tumor suppressor genes ( p 53 , rb , mts ) involved in lung cancer development with respect to the pathology of lung tumors , including the importance of these genes as far as the clinical course of the disease is concerned . . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
DNA probes for the NRAS , HRAS , KRAS 2 , LCK , RAF 1 , MET , MYCL 1 , MYCN , MYB , ERBB 2 , FOS , CSF1R , and SRC protooncogene loci ; the retinoblastoma gene locus ( RB 1 ) ; the tumor virus integration sites INT 2 , PVT 1 , and MLV 12 ; and the locus of the tumor specific antigen T1A were used to screen mouse genomic DNAs from RF / J , CAST / Ei , MOLF / Ei , Mus musculus musculus , M . m . poschiavinus , and M . spretus . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
RESULTS : In contrast to non endocrine tumours of the digestive system such as carcinoma of the pancreas , colon and stomach , dNETs do not show alterations in oncogenes ( ras , Myc , fos jun , Src ) or in common tumor suppressor genes [ p 53 , retinoblastoma suspectibility gene ( Rb ) ] . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
Among the genes that were differentially regulated were fos , RB 1 , glutathione peroxidase , TGF beta , and 15 lipoxygenase , a gene known to be activated by mutant p 53 . ^^^
Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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Interacting proteins: P01100 and P06400	Pubmed	SVM Score :0.0
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