| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| NA |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| Adhesion of hepatocytes to the EHS gel for the same period of time dramatically alters this program : it arrests growth and inhibits AP 1 DNA binding activity and the expression of c jun , junB , and c myc mRNAs , but , in addition , it restores C / EBP alpha mRNA and protein as well as C / EBP alpha and C / EBP beta DNA binding activities to the abundant levels present in freshly isolated hepatocytes . ^^^ |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| These substrates include transcription factors that are regulated by MAP kinase phosphorylation ( e . g . , Elk 1 , c Myc , c Jun , c Fos , and C / EBP beta ) . ^^^ |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| This induction of PEPCK expression by cAMP occurred in the presence of sustained levels of CCAAT / enhancer binding protein ( C / EBP ) alpha delta mRNAs , and was accompanied by an increase in the rate of transcription and mRNA content of C / EBP beta gene , and a decrease in the expression of c myc , in the absence of c fos expression . ^^^ |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| Cell transformation by the 5 myc oncogene abrogates c Myc / Max mediated suppression of a C / EBP beta dependent lipocalin gene . ^^^ The promoter region contains consensus binding sites for the transcriptional regulators Myc and C / EBP beta . ^^^ Transcriptional activation of Q 83 is principally dependent on C / EBP beta , but is blocked in normal cells by the endogenous c Myc / Max / Mad transcription factor network . ^^^ In 5 myc transformed cells , high level expression of the 5 Myc protein and formation of highly stable 5 Myc / Max heterodimers leads to abrogation of Q 83 gene suppression and activation by C / EBP beta . ^^^ |
|
| Interacting proteins: P17676 and P01106 |
Pubmed |
SVM Score :0.0 |
| These markers include molecules known to be important in conventional gastric carcinogenesis , such as E Cadherin , p 53 , COX 2 , Trefoil Factor 1 ( TFF 1 ) , beta catenin , p 16 and c myc ; as well as molecules not yet described as being important in gastric cancer , such as the transcription factor c jun , the COX 2 mRNA stabilizer HuR , and C / EBP beta , a transcription factor for COX 2 . ^^^ |
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